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On the other hand, with inconclusive research, taurine might be useful to the balance in between the cytokine response and the anti-inflammatory response following exercise [69]. Taurine concentrations in cardiac mitochondria are estimated in high concentrations to be 70 nmoL/mg [26]). It has been suggested that higher concentrations of taurine in the cardiac mitochondria can inhibit mitochondrial apoptosis, oxidative and endoplasmic reticulum strain, and serve as a mitochondrial buffer [34]. The acyl-CoA dehydrogenases which handle the β-oxidation of fatty acids are shown to have optimal activity with mitochondrial taurine serving as a mitochondrial buffering agent [26]. For example Sports Gamble, the rate of β-oxidation of endogenous fatty acids was 31% reduced in the taurine-deficient heart in mice compared to the handle heart [65]. Stabilizing the pH gradient in the mitochondrial matrix by the presence of taurine as a buffer is essential for maintaining the biochemical processes. Taurine has also been demonstrated to be a constituent of modified uridine residues in mitochondrial tRNA [26]. The modified uridine residues in the mitochondria suggest that taurine might play a part in the mitochondrial matrix and be implicated in mitochondrial ailments such as cardiovascular illness.